Embryonic Anoxia Alters Exploratory Behavior in Juvenile Geckos

Presentation Type

Poster

Full Name of Faculty Mentor

Ryan Yoder, Psychology

Major

Psychology

Presentation Abstract

Environmental changes, such as temporary anoxia, during the embryonic stage can impair brain development in leopard geckos (Eublepharis macularius). We therefore tested whether this early brain damage produces persistent behavioral deficits. The organization and kinematic properties of non-visual exploration between normal (n = 5) and anoxia (n = 4) geckos were compared. Geckos were individually placed on a circular table (diam=91cm) and allowed to explore dark conditions for 60 min while being recorded. The gecko’s coordinates were calculated at 3 frames/second. Movement properties within each trial were evaluated across five 10 min epochs. Total distance, peak speed, movement scaling (correlation between path length and peak speed), distance ratio, heading error, total stop time, mean stop time, number of stops, number of progressions, and progression distance were compared between groups and across epochs with a mixed Group X Epoch ANOVA. We found that total stop time and overall number of stops were significantly lower in anoxia geckos. These results could indicate that brain damage caused by early anoxia causes geckos to be able to process less information, which would lead to less overall stops and decreased stop length. Geckos retain the ability to move normally and accurately estimate distance. Thus, the reduced stops are not caused by a general movement deficit. These preliminary results suggest that embryonic anoxia persistently alters exploratory behavior. This on-going study will continue to evaluate exploratory movement, and data will be added to the presentation as they become available.

Start Date

12-4-2023 4:00 PM

End Date

12-4-2023 6:00 PM

Disciplines

Psychology

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Apr 12th, 4:00 PM Apr 12th, 6:00 PM

Embryonic Anoxia Alters Exploratory Behavior in Juvenile Geckos

Environmental changes, such as temporary anoxia, during the embryonic stage can impair brain development in leopard geckos (Eublepharis macularius). We therefore tested whether this early brain damage produces persistent behavioral deficits. The organization and kinematic properties of non-visual exploration between normal (n = 5) and anoxia (n = 4) geckos were compared. Geckos were individually placed on a circular table (diam=91cm) and allowed to explore dark conditions for 60 min while being recorded. The gecko’s coordinates were calculated at 3 frames/second. Movement properties within each trial were evaluated across five 10 min epochs. Total distance, peak speed, movement scaling (correlation between path length and peak speed), distance ratio, heading error, total stop time, mean stop time, number of stops, number of progressions, and progression distance were compared between groups and across epochs with a mixed Group X Epoch ANOVA. We found that total stop time and overall number of stops were significantly lower in anoxia geckos. These results could indicate that brain damage caused by early anoxia causes geckos to be able to process less information, which would lead to less overall stops and decreased stop length. Geckos retain the ability to move normally and accurately estimate distance. Thus, the reduced stops are not caused by a general movement deficit. These preliminary results suggest that embryonic anoxia persistently alters exploratory behavior. This on-going study will continue to evaluate exploratory movement, and data will be added to the presentation as they become available.